Seborrhoeic dermatitis

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Seborrhoeic dermatitis
Classification & external resources
ICD-10 L21.
ICD-9 690
DiseasesDB 11911
MedlinePlus 000963
eMedicine derm/396 
MeSH D012628

Seborrheic dermatitis is a skin disorder affecting the scalp, face and trunk causing scaly, flaky, itchy, red skin. It particularly affects the sebum-gland rich areas of skin.

Contents

The cause of seborrheic dermatitis remains unknown, although many factors have been implicated. The ubiquitous yeast, Malassezia furfur (formerly known as Pityrosporum ovale), is involved,[1][2] as well as genetic, environmental, hormonal, and immune-system factors. [3][4] A suggestion that seborrheic dermatitis is an inflammatory response to this yeast has yet to be proven.[5] Those afflicted with seborrheic dermatitis have an unfavorable epidermic response to the infection, with the skin becoming inflamed and flaking.

Side effects to inflammation may include temporary hair loss. Note that if severe outbreaks go untreated for long periods of time, permanent hair loss may result due to damaged hair follicles.

Expect two to six months before hair growth may resume.

Soaps and detergents such as Sodium Laureth Sulfate may precipitate a flare-up, as they strip moisture from the top layers of the skin, and the drying property of these can cause flare-ups and may worsen the condition.[citation needed] Accordingly a suitable alternative should be used instead.

Among dermatologist recommended treatments are shampoos containing coal tar, ciclopiroxolamine ketoconazole, selenium sulfide, or zinc pyrithione.[6] For severe disease, keratolytics such as salicylic acid or coal tar preparations may be used to remove dense scale. Topical terbinafine solution (1%) has also been shown to be effective in the treatment of scalp seborrhea,[7] as may lotions containing alpha hydroxy acids or corticosteroids. Pimecrolimus topical lotion is also sometimes prescribed.

Chronic treatment with topical corticosteroids may lead to permanent skin changes, such as atrophy and telangiectasia.[8][9]

UV-A and UV-B light inhibit the growth of M. furfur[10], although caution should be taken to avoid sun damage.

Those with seborrheic dermatitis might benefit from biotin supplements. One might also try a humidifier by the bed, as well as a gentle moisturizer with or without oatmeal.

  1. ^ Hay R, Graham-Brown R (1997). "Dandruff and seborrheic dermatitis: causes and management". Clin Exp Dermatol 22 (1): 3-6. PMID 9330043. 
  2. ^ Nowicki R (2006). "[Modern management of dandruff]". Pol Merkur Lekarski 20 (115): 121-4. PMID 16617752. 
  3. ^ Am Fam Physician 2000;61:2703-10,2713-4
  4. ^ Janniger C, Schwartz R (1995). "Seborrheic dermatitis". Am Fam Physician 52 (1): 149-55, 159-60. PMID 7604759. 
  5. ^ Parry M, Sharpe G (1998). "Seborrheic dermatitis is not caused by an altered immune response to Malassezia yeast". Br J Dermatol 139 (2): 254-63. PMID 9767239. 
  6. ^ Schwartz R, Janusz C, Janniger C (2006). "Seborrheic dermatitis: an overview". Am Fam Physician 74 (1): 125-30. PMID 16848386. 
  7. ^ Faergemann J, Jones J, Hettler O, Loria Y (Jun 1996). "Pityrosporum ovale (Malassezia furfur) as the causative agent of seborrheic dermatitis: new treatment options". Br J Dermatol 134 Suppl 46: 12-5: discussion 38. PMID 8763461. 
  8. ^ Smith J, Wehr R, Chalker D (1976). "Corticosteroid-induced cutaneous atrophy and telangiectasia. Experimental production associated with weight loss in rats". Arch Dermatol 112 (8): 1115-7. PMID 952530. 
  9. ^ Scheinfeld N (2005). "Seborrheic dermatitis". Skinmed 4 (1): 49-50. PMID 15654167. 
  10. ^ Wikler J, Janssen N, Bruynzeel D, Nieboer C (1990). "The effect of UV-light on pityrosporum yeasts: ultrastructural changes and inhibition of growth". Acta Derm Venereol 70 (1): 69-71. PMID 1967880. 

Seborrheic dermatitis information and natural treatment http://health.groups.yahoo.com/group/seborrheic-derma/

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